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Glucose and oleic acid mediate cellular alterations in GLP‐1‐induced insulin‐positive differentiating UCBMSCs | Request PDF
LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
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PDF) Deciphering the Molecular Role of Non-coding RNA in Malignant Cardiac Disease | published in Biointerface Research in Applied Chemistry
LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
Processes | Free Full-Text | Competing Endogenous RNAs in Cervical Carcinogenesis: A New Layer of Complexity | HTML
Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
Modification of miRNA Expression through plant extracts and compounds against breast cancer: Mechanism and translational significance - ScienceDirect
LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
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IJERPH | Free Full-Text | Exposomes to Exosomes: Exosomes as Tools to Study Epigenetic Adaptive Mechanisms in High-Altitude Humans | HTML
LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
Frontiers | Endoplasmic Reticulum-Mitochondria Contacts: A Potential Therapy Target for Cardiovascular Remodeling-Associated Diseases
VORTEX | Racing Kart - leader store in the kart world
VORTEX | Racing Kart - leader store in the kart world
Processes | Free Full-Text | Competing Endogenous RNAs in Cervical Carcinogenesis: A New Layer of Complexity | HTML
LncRNA NEAT1 knockdown ameliorates LPS-induced human kidney injury by mediating the miR-330-5p/FOXO3 axis | SpringerLink
IJERPH | Free Full-Text | Exposomes to Exosomes: Exosomes as Tools to Study Epigenetic Adaptive Mechanisms in High-Altitude Humans | HTML
Reappraisal of metabolic dysfunction in neurodegeneration: Focus on mitochondrial function and calcium signaling
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Reappraisal of metabolic dysfunction in neurodegeneration: Focus on mitochondrial function and calcium signaling
Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
Lockdown of mitochondrial Ca2+ extrusion and subsequent resveratrol treatment kill HeLa cells by Ca2+ overload - ScienceDirect
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